Last night was one of those nights when I could not easily fall asleep.  I’m conducting an experiment.  I’m feeling compelled to sleep on my back to see if over six months my left-side, 12-13 mm quasi-fusiform carotid artery cerebral aneurysm diminishes in size.  I’ve slept on my right side my whole life.  It’s not likely, based on what the surgeons have suggested, that sleeping on my back can have an effect.  Still, I feel like there is something I can do to make a difference.  The night I came to this conclusion was the night that the four-pole thesis began to form.

So I sleep light instead of my customary deep.  Mortality feels not particularly far away.  Often my mind starts wrestling with the principles discussed in this blog.  On nights like last night, I move the concepts around a 4-D space, looking for new relationships.  A couple things came to mind last night.

In the four-pole hypothesis….

F te/M TE        Conventional Patrifocal
F tE/M Te        Warrior Patrifocal
F Te/M tE        Contemporary Matrifocal
F TE/M te        Classic Matrifocal

….  I’d anticipate that representatives of all four pairings, all eight individual prototypes, would be represented in most societies.  Very few societies would skew toward the matrifocal in our times, but perhaps, based on handedness studies, West African societies such as the Noruba would skew left.  Balanced polymorphisms would vary from society to society; most people would not exhibit extreme representatives of the eight prototypes, but those that did would likely make up a far higher percentage of the disease, condition, disorder population than the folks tending toward the middle of the curve.

Geschwind and Galaburda (1987) suggested that the anomalously dominant cerebral hemisphere community would exhibit both increases in maladies (such as asthma) and increases in mental and physiological gifts (such as mathematics and athletic skills).  With our four-pole hypothesis, we might come to the same conclusion, particularly because it is the same model with the addition of estrogen, including perspectives derived from anthropology (social structure) and evolutionary biology (heterochronic theory).

Additional insights come into play.

We would hypothesize that the eight hormonal constellations exhibited by the eight individual prototypes in the four-pole hypothesis are staking out eight disease, disorder and condition regions.  These being the hormonal constellation boundaries of social structure, we’d expect that here would emerge any number of clashes between hormone-driven somatic structure and the places where physical structure ceases to perform.  This is where the rubber meets the road in evolution.  This is where the limits of what a body can hormonally endure is exposed to conditions unfriendly to seamless functioning, the extremes of the balanced polymorphism.  And, as Geschwind, Annett and their colleagues have hypothesized, here we’ll also find examples of the gifted.

Consider that a disease, condition or disorder may appear in more than one of the eight prototype humans.  Annett noted that there are two kinds of dyslexia, evidencing two different kinds of verbal difficulty.  One was associated with anomalous dominance.  The other was associated with extreme right-handers.  It is possible that with this eight-prototype theory, other conditions will be discovered to have more than one etiology.  Autism was provided as an example in the last entry.

This is the stuff I was playing with in my head last night.  Unable to sleep, I was seeking to understand “dis-ease.”


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